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Palpitations
What are palpitations?
Palpitations are unpleasant sensations of irregular
and/or forceful beating of the heart. Some persons with
palpitations have no heart disease or abnormal heart
rhythms and the reasons for their palpitations are
unknown. In others, palpitations result from abnormal heart rhythms
(arrhythmias).
- Arrhythmias refer to heartbeats that are
too slow, too rapid, irregular, or too early.
- Rapid arrhythmias (greater than 100 beats per
minute) are called tachycardias.
- Slow arrhythmias (slower than 60 beats per minute)
are called bradycardias.
- Irregular heart rhythms are called
fibrillations (as in atrial
fibrillation).
- When a single heartbeat occurs earlier than
normal, it is called a premature contraction,
and this can cause the sensation of a forceful
heartbeat.
- Abnormalities in the atria,
the ventricles, and the electrical conducting system
[the Sino-atrial (SA) node, and the Atrio-ventricular
(AV) node] of the heart can lead to arrhythmias that
cause palpitations.
How are palpitations
evaluated?
The first step in the evaluation of
patients with palpitations is to determine whether their
symptoms are actually due to arrhythmias. Because the
treatment of varying types of arrhythmias can differ, it
is also important to determine the type of arrhythmias
involved. Since arrhythmias can be related to underlying
disease of the heart valves, heart muscle, and coronary
arteries; tests are often performed to exclude heart
abnormalities. Blood tests are also obtained to measure
blood sodium, potassium, calcium, magnesium, thyroid
hormone levels, and medication levels (such as digoxin
levels).
Tests for arrhythmias include
resting electrocardiogram
(EKG), 24-hour rhythm monitoring (Holter), and a
treadmill exercise
test.
A resting EKG is a short recording
of the heart's electrical activity, usually performed in
the doctor's office. An EKG is useful only if the
arrhythmia causing the palpitations is occurring when
the EKG is being recorded. Often, the resting EKG cannot
catch the arrhythmias, and a 24 hour Holter monitor is
required. The 24 hour Holter is a cassette tape worn by
the patient continuously while carrying out his/her
usual activities. The patient simultaneously keeps a
diary of palpitations or other symptoms during the
recording period. Symptoms of palpitations can later be
correlated with the presence or absence of arrhythmias
on the Holter tape. If suspected arrhythmias causing
palpitations still cannot be captured by the 24 hour
Holter, a small patient-activated event monitor is worn
by the patient for 1 to 2 weeks. When the patient
experiences palpitation, he/she presses a button to
record the heart rhythm prior to, during, and after the
episode. The recordings can be analyzed by a doctor at a
later date.
In some patients, exercise treadmill
is used to detect arrhythmias that occur only with
exertion. Exercise treadmill is a continuous EKG
recording of the heart as the patient performs
increasing levels of exercise. In addition to detecting
arrhythmias, exercise treadmill is a useful screening
test for the presence of narrowed coronary arteries that
can limit supply of oxygenated blood to the heart muscle
during exercise.
Echocardiography uses ultrasound waves to obtain images of the heart
chambers, valves and surrounding structures.
Echocardiography is useful in detecting diseases of the
heart valves, such as mitral valve prolapse, mitral
stenosis, and aortic stenosis
(examples of valve diseases that can cause arrhythmias
and palpitations). Echocardiography is also useful in
evaluating the size of heart chambers, as well as the
health and contractions of the muscle of the ventricles.
Combining echocardiography with exercise stress testing
(stress echocardiography) is an accurate screening test
for significant coronary artery disease. The portion of
the ventricles supplied by the narrowed arteries does
not contract as well as the rest of the ventricles
during exercise.
Occasionally, cardiac
catheterization with angiography
is performed to detect disease in the coronary arteries
or in the heart valves which is triggering an
arrhythmia. Coronary arteries supply oxygenated blood
from the aorta to the heart muscle. During this
procedure, a small hollow plastic tube is advanced under
x-ray guidance from an artery in the groin to the
openings of the two main coronary arteries located above
the aortic valve. A contrast agent, made of iodine, is
then injected into the arteries while x-ray pictures are
recorded. It is an accurate test in detecting, mapping,
and assessing the extent and severity of coronary artery
disease. For further information, please read the
Angina, and Angioplasty
articles.
Blood tests are performed to measure
the levels of thyroid hormone, potassium, magnesium, and
medications, such as digoxin. Excess thyroid hormone can
lead to rapid arrhythmias, such as atrial fibrillation.
Low blood levels of potassium and magnesium can lead to
other arrhythmias. Digoxin (Lanoxin) toxicity can cause
serious arrhythmias, such as bradycardia, and
ventricular tachycardias. Digoxin toxicity can be
enhanced by low blood levels of potassium and
magnesium.
How are
palpitations managed?
Palpitations without
associated arrhythmias and heart diseases may not
require specific treatment. Patients are often advised
simply to reduce emotional and physical stress while
monitoring their symptoms.
Palpitations resulting from
premature contractions (PACs and PVCs) often require no
specific treatment. The frequency of premature
contractions can be reduced by stress reduction,
quitting smoking, and reducing caffeine, and alcohol
consumption. High blood adrenaline levels can lead to
premature contractions, while stress reduction helps to
lower blood adrenaline levels. For patients with
persistent palpitations and premature contractions,
medications, such as beta-blockers, can be used to block
the effect of adrenaline on the heart, thus reducing
premature contractions. Examples of beta-blockers
include propranolol (Inderal), metoprolol (Lopressor), and atenolol (Tenormin). Side effects of
beta
blockers include
worsening of asthma,
excessive lowering of heart rate and blood pressure,
depression, fatigue, and impotence.

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